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A peer-reviewed Jan. 2012 study in the Journal of Neuro-Oncology concluded that RF radiation "may damage DNA and change gene expression in brain cells" in mice.  An Aug. 2009 meta-study found that RF radiation "can alter the genetic material of exposed cells."  A 2004 European Union-funded study also found that cell phone radiation can damage genes.  On May 26, 2016, the US National Toxicology Program (NTP) released the first results of its study on cell phone radiation, finding an increased incidence of malignant tumors of the brain (gliomas) and heart tumors (schwannomas) in rats exposed to RF radiation.  The NTP researchers also found DNA damage in the rats exposed to the highest levels of RF radiation.  On Nov. 1, 2018, the NTP released its final peer-reviewed report, concluding that there is "clear evidence of carcinogenic activity” in male rats exposed to RF radiation. 
Repeated Wi-Fi studies show that Wi-Fi causes oxidative stress, sperm/testicular damage, neuropsychiatric effects including EEG changes, apoptosis, cellular DNA damage, endocrine changes, and calcium overload. Each of these effects are also caused by exposures to other microwave frequency EMFs, with each such effect being documented in from 10 to 16 reviews. Therefore, each of these seven EMF effects are established effects of Wi-Fi and of other microwave frequency EMFs. Each of these seven is also produced by downstream effects of the main action of such EMFs, voltage-gated calcium channel (VGCC) activation. While VGCC activation via EMF interaction with the VGCC voltage sensor seems to be the predominant mechanism of action of EMFs, other mechanisms appear to have minor roles. Minor roles include activation of other voltage-gated ion channels, calcium cyclotron resonance and the geomagnetic magnetoreception mechanism.
These general findings and data presented earlier on Wi-Fi effects were used to assess the Foster and Moulder (F&M) review of Wi-Fi. The F&M study claimed that there were seven important studies of Wi-Fi that each showed no effect. However, none of these were Wi-Fi studies, with each differing from genuine Wi-Fi in three distinct ways. F&M could, at most conclude that there was no statistically significant evidence of an effect. The tiny numbers studied in each of these seven F&M-linked studies show that each of them lack power to make any substantive conclusions.
The advent of Wi-Fi connected high technology devices in executing day-to-day activities is fast evolving especially in developing countries of the world and hence the need to assess its safety among others. The present study was conducted to investigate the injurious effect of radiofrequency emissions from installed Wi-Fi devices in brains of young male rats. Animals were divided into four equal groups; group 1 served as control while groups 2, 3, and 4 were exposed to 2.5 Ghz at intervals of 30, 45, and 60 consecutive days with free access to food and water ad libitum. Alterations in harvested brain tissues were confirmed by histopathological analyses which showed vascular congestion and DNA damage in the brain was assayed using agarose gel electrophoresis. Histomorphometry analyses of their brain tissues showed perivascular congestion and tissue damage as well.
The present study tested the effects of Wi-Fi (2.45 GHz for 1h) exposure on Ca(2+) influx, oxidative stress and apoptosis through TRPV1 channel in the murine dorsal root ganglion (DRG) and hippocampus of pentylentetrazol (PTZ)-induced epileptic rats. The cytosolic free Ca(2+), reactive oxygen species production, apoptosis, mitochondrial membrane depolarization, caspase-3 and -9 values in hippocampus were higher in the PTZ group than in the control although cell viability values decreased. The Wi-Fi exposure induced additional effects on the cytosolic Ca(2+) increase. However, pretreatment of the neurons with CPZ, results in a protection against epilepsy-induced Ca(2+) influx, apoptosis and oxidative damages. In conclusion, epilepsy and Wi-Fi in our experimental model is involved in Ca(2+) influx and oxidative stress-induced hippocampal and DRG death through activation of TRPV1 channels, and negative modulation of this channel activity by CPZ pretreatment may account for the neuroprotective activity against oxidative stress.
A method of simulating thermal mapping of positioning laptop on laps of an adult man was developed. To tackle this problem, we exploited computer simulation and, to make the simulation close to the actual problem, we created 3D models of an actual laptop (Sony FW 590 Gab), antennas, and human phantom with inhomogeneous body, large number of tissues, and dispersion properties. We employed a commercial laptop Wi-Fi antenna at 2.4 GHz and a dipole antenna at 5 GHz, thermal sources with radiation powers, and human body voxel consisting of 97 tissues which were described previously. In the simulation, maximum SAR in human body was calculated 0.37 × 10−3 and 0.13 × 10−1 (W/kg) at 2.4 and 5 GHz, respectively, which was negligible according to IEEE standards; thus, the major calculated temperature elevation was due to laptop thermal sources. The temperature in glans penis, lap skin, lap muscles, and testes increased up to 37.8, 42.9, 38.8, and 37.2 °C, respectively, which was in line with clinical studies of thermal effect. Hence, the proposed method can be replicated for other scenarios. It is worth noting that the presented result cannot be easily generalized to other devices or human models. However, the whole method is replicable for similar phenomena. The recommended subject for future works can be used with the presented method for determining the effect of laptop and other devices on adult pregnant women and similar cases.
Another animal study, in which rats were exposed 7 days per week for 19 hours per day to radiofrequency radiation at 0.001, 0.03, and 0.1 watts per kilogram of body weight was reported by investigators at the Italian Ramazzini Institute (35). Among the rats with the highest exposure levels, the researchers noted an increase in heart schwannomas in male rats and non-malignant Schwann cell growth in the heart in male and female rats. However, key details necessary for interpretation of the results were missing: exposure methods, other standard operating procedures, and nutritional/feeding aspects. The gaps in the report from the study raise questions that have not been resolved.
Cellsafe have developed revolutionary NEW Smart Radi-Chip technology, giving you the freedom to use any protective phone / iPad case or to enjoy your device without a case while reducing radiation absorption by up to 95%* (* refer to test results page for individual results according to phone make & frequency band). This product is micro-thin (0.35mm) adhesive chip which attaches to the back of the phone / iPad and can be used with any mobile phone / iPad case fitted over the top. Suitable for most popular Smart Phones & iPads. Cellsafe’s groundbreaking technology is NOT a shield and will not interfere with transmission signal or with call reception.
According to the results of the current research, long-term exposure to EMR emitted from wireless devices had adverse effects on the antioxidant potential of blood. Therefore, to protect individuals from harmful effects of Wi-Fi signals, it is advised to limit the use of such devices for household and occupational activities, if possible. This study may stimulate future helpful research in the development of new protective or therapeutic approaches. In addition, it is recommended that the target tissues of EMR emitted from wireless devices and the level of other mediators be investigated to understand the exact molecular mechanism and site of action upon continuous exposure to such radiations.
Numerous peer-reviewed studies have found that cell phone use is not associated with an increased risk of brain tumors. An Oct. 20, 2011 study of 358,403 Danish citizens – the largest study of its kind to date – concluded that "there was no association between tumors of the central nervous system or brain and long term (10 years +) use of mobile phones."  A July 27, 2011 study found that there was no association between cell phone use and brain tumor risks among children and adolescents.  Numerous other studies published from 2001-2013 have similarly concluded that there is no association between cell phone use and the development of brain tumors.      
The present study was designed to determine the effects of 2.45 GHz radiation (1, 2, 12, or 24 hours) on the antioxidant redox system, calcium ion signaling, cell count and viability in human leukemia 60 cells.The extent of lipid peroxidation, cytosolic free Ca²⁺ and cell numbers were higher in 2.45 GHz groups than in the controls and was time-dependent. 2.45 GHz electromagnetic radiation appears to induce proliferative effects through oxidative stress and Ca²⁺ influx although blocking of transient receptor potential melastatin 2 channels by 2-aminoethyl diphenylborinate seems to counteract the effects on Ca²⁺ ions influx.
This study aimed to examine the therapeutic effects of a 100 Hz pulsed electromagnetic field (2 h/day for 60 days) on the reproductive systems of male Wistar rats (70 days old).The results showed significant increases in caspase and creatine kinase and significant decreases in testosterone and melatonin in the exposed groups. This finding emphasizes that reactive oxygen species (a potential inducer of cancer) are the primary cause of DNA damage. However, pulsed electromagnetic field exposure relieves the effect of microwave exposure by inducing Faraday currents.
The HARApad laptop pad is available in 3 sizes and multiple colors. There's even an attractive wood option, which allows for a beautiful, natural approach to laptop radiation shielding. There are size options for 13″, 15″ and 17″ laptops, and even a 25″ LapDesk version that will allow for the convenient use of a mouse, if you use a mouse instead of a trackpad.
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