The revolving-door syndrome that characterizes so many industries and federal agencies reinforces the close relationship between the wireless industry and the FCC. Just as Tom Wheeler went from running the CTIA (1992– 2004) to chairing the FCC (2013–2017), Meredith Atwell Baker went from FCC commissioner (2009–2011) to the presidency of the CTIA (2014 through today). To ensure its access on Capitol Hill, the wireless industry made $26 million in campaign contributions in 2016, according to the Center for Responsive Politics, and spent $87 million on lobbying in 2017.32

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The present study aimed to investigate the protective effects of melatonin against oxidative stress-mediated testicular impairment due to long-term exposure (2 h/day for 45 days) of 2.45 GHz Microwave Radiation. Result shows that melatonin prevented oxidative damage biochemically by significant increase (p < 0.001) in the levels of testicular LDH-X, decreased (p < 0.001) levels of MDA and ROS in testis (p < 0.01). Meanwhile, it reversed the effects of MWs on XO, protein carbonyl content, sperm count, testosterone level and DNA fragmentation in testicular cells. These results concluded that the melatonin has strong antioxidative potential against MW induced oxidative stress mediated DNA damage in testicular cells.
The present study was undertaken to determine the influence of low intensity microwave radiation (900, 1800, or 2450 MHz for 2h/day, 5days/week, for 60 days) on oxidative stress, inflammatory response and DNA damage in rat brain. Low intensity microwave exposure resulted in a frequency dependent significant increase in oxidative stress markers, reduced levels of GSH and SOD, increased levels of pro-inflammatory cytokines, and significant DNA damage in microwave exposed groups compared to controls. In conclusion, the present study suggests that low intensity microwave radiation induces oxidative stress, inflammatory response and DNA damage in brain by exerting a frequency dependent effect. The study also indicates that increased oxidative stress and inflammatory response might be the factors involved in DNA damage following low intensity microwave exposure.

Several studies that will provide more information are under way. Researchers from the Centre for Research in Environmental Epidemiology in Spain are conducting another international case–control study—Mobi-Kids—that will include 2000 young people (aged 10–24 years) with newly diagnosed brain tumors and 4000 healthy young people. The goal of the study is to learn more about risk factors for childhood brain tumors.

If cell phones were causing cancer we could expect a significant rise in the rate of brain and other related cancers. According to the National Cancer Institute, there was no increase in the incidence of brain or other nervous system cancers between the years 1987 and 2005 despite the fact that cell phone use dramatically increased during those same years. [6] Between 2004 and 2010 there was still no significant change in the incidence rate of brain tumors. Between 2004 and 2010 there was a slight increase from 209 cases to 221.8 cases per 100,000 people, but this slight increase was attributed to better tracking and recording of cases. [43] During the same time period, cell phone use increased 62.7% from 182,140,362 subscribers in 2004 to 296,285,629 in 2010. [44]

BACKGROUND: Dental amalgam is composed of approximately 50% elemental mercury. Despite concerns over the toxicity of mercury, amalgam is still the most widely used restorative material. Wi-Fi is a rapidly using local area wireless computer networking technology. To the best of our knowledge, this is the first study that evaluates the effect of exposure to Wi-Fi signals on mercury release from amalgam restorations.
Participation bias, which can happen when people who are diagnosed with brain tumors are more likely than healthy people (known as controls) to enroll in a research study. Also, controls who did not or rarely used cell phones were less likely to participate in the Interphone study than controls who used cell phones regularly. For example, the Interphone study reported participation rates of 78% for meningioma patients (range among the individual studies 56–92%), 64% for glioma patients (range 36–92%), and 53% for control subjects (range 42–74%) (6).
Remarkably, cell phones had been allowed onto the US consumer market a decade earlier without any government safety testing. Now, some customers and industry workers were being diagnosed with cancer. In January 1993, David Reynard sued the NEC America Company, claiming that his wife’s NEC phone caused her lethal brain tumor. After Reynard appeared on national TV, the story went viral. A congressional subcommittee announced an investigation; investors began dumping their cell-phone stocks; and Wheeler and the CTIA swung into action.2
Depends on if you have other sources present in your home as well. Cordless home phone+base station, Smart Meter, Non-eco Wifi-Router (Eco will reduce the 10 pulses to 1 pulse per second on your Router, even when it has no connected devices.) An ethernetcable will work great, but make sure to disable the active wifi connection on your Laptop or Pc.

We aimed to investigate the effects of distance from sources on calcium signaling, cytosolic ROS production, cell viability, apoptosis, plus caspase-3 and -9 values induced by mobile phones and Wi-Fi in breast cancer cells. The cytosolic ROS production, Ca2+ concentrations, apoptosis, caspase-3 and caspase-9 values were higher in groups exposed to 900 MHz, 1800 MHz and 2450 MHz compared to controls at 0 cm, 1 cm and 5 cm distances although cell viability (MTT) values were increased by the distances. There was no statistically significant difference in the values between control, 20 and 25 cm. Wi-Fi and mobile phone EMR placed within 10 cm of the cells induced excessive oxidative responses and apoptosis via TRPV1-induced cytosolic Ca2+ accumulation in the cancer cells. Using cell phones and Wi-Fi sources which are farther away than 10 cm may provide useful protection against oxidative stress, apoptosis and overload of intracellular Ca2+.
Belgium recently adopted new cell phone regulations that bar mobile phone models designed for, and marketed to children ages 7 and younger.  Under Belgium’s new rules, slated to take effect next March, cell phone retailers will be required to disclose phones’ maximum emission levels, known as specific absorption rates, or SAR, at the point of sale.  Belgium becomes the latest in a rapidly lengthening list of nations to attempt to shield children from too much cell phone radiation and to inform everyone about the risks of exposure to these emissions. At least a dozen other nations have taken steps to protect children from cell phone radiation.
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Since children’s skulls are thinner than adults’ and their nervous systems are still developing, researchers believe they may be at a greater risk for cell phone-related cancers. They’ll also be exposed to the potentially harmful radiation significantly longer than their parents’ generation. Until more is known about the possible carcinogenic effects of cell phone use, curb your kids’ cell phone habits by encouraging texting or using a landline instead—if they can actually recognize a landline anymore, that is.
Anything you can do to keep the phone away from your head will help. (Even cell phone manufacturers warn consumers about this. See the previous post Cell Phone Manual Warnings for examples of warnings from Samsung, Apple and more.) If you hold the phone 2 inches away, the signal is about one-fourth the original strength. At four inches away, it’s about 1/16th as strong. Even better is a hands-free kit with a wireless air tube nearest the earpiece. (A regular wired headset can act as an antenna and make matters worse.)
This study elucidates the effect of short (15 days) and long-term (30 and 60 days) low level 2.45 GHz MW radiation induced local stress on the hippocampal spatial memory formation pathway in adult male mice. Researchers observed that 2.45 GHz MW irradiated mice showed slow learning and significantly increased number of working and reference memory errors in RAM task. Further, 2.45 GHz MW radiation exposure increases serum corticosterone level and the expression of CRH, CRH-R1 and i-NOS, while the expression of iGluRs, n-NOS, PSD-95, PKCε, PKA, ERK-p-ERK, CREB and p-CREB decreases in above mentioned hippocampal subregions in a duration dependent manner. “Our findings led us to conclude that 2.45 GHz MW radiation exposure induced local stress suppresses signaling mechanism(s) of hippocampal memory formation.”
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In a separate study by the same Swedish team, they found more than seven times the risk among people using a cell phone more than 20 years and 6.5 times the risk for long-term users of cordless phones. As expected, most of the gliomas and acoustic neuromas were on the same side of the head, which was usually exposed to the phone. In the 2013 official report on the medical evidence for brain tumors, the International Agency for Research on Cancer concluded that radiation from cell phones is “possibly carcinogenic to humans”.