Unfortunately, the current state of cell phone radiation research is similar to the early days of tobacco research. It wasn't until 1999 when the U.S. Department of Justice finally filed a racketeering lawsuit against the major tobacco companies for engaging in a "50 year scheme" to counteract any scientific evidence that showed that cigarettes are harmful to a person's health. Is the potential cell phone cancer link the same sort of scenario? Are cell phone companies funding and promoting research that "proves" no link from cell phone radio radiation in an effort to discredit those studies that are uncovering problems?
DefenderShield Cellphone Radiation Case also claims independent testing and says in their website: That a "sophisticated layering of separate non-toxic, human safe materials processed for maximum radiation blocking efficiency. Each material has unique and targeted radiation-shielding characteristics designed to work in unison to eliminate all radiation emissions from 0 to 10 GHZ and Defender Shield technology refracts, conducts and finally absorbs all these potentially harmful emissions." In this demonstration, the radiation level measured when DefendeShield case was on is still quite a bit above the level of exposure that I would recommend. I personally do not recommend-holding a phone with a shielding case to your head as the DefenderShield website shows a young woman doing as she demonstrates the product.
A phone's specific absorption rate (SAR) reveals the maximum amount of radiation the human body absorbs from the phone while it's transmitting. SAR testing ensures that the devices sold in the U.S. comply with the Federal Communications Commission (FCC) SAR exposure limit, but the single, worst-case value obtained from this SAR testing is not necessarily representative of the absorption during actual use, and therefore it is not recommended for comparisons among phones. In short, selecting a lower SAR phone will not reliably ensure lower radiation absorption during use. The FCC has more information at Specific Absorption Rate (SAR) For Cell Phones: What It Means For You.
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This study investigated the effect of 2GHz EMR (1h) on the growth dynamics of Myriophyllum aquaticum (Parrot feather) by measuring the nanometric elongation rate fluctuation (NERF) using a statistical interferometry technique. After continuous exposure to EMR, M. aquaticum plants exhibited a statistically significant reduction in NERF standard deviation, therefore, the reduced NERF was due to a non-thermal effect caused by EMR exposure. The alteration in NERF continued for at least 2.5 h after EMR exposure and no significant recovery was found in post-EMR NERF during the experimental period.
The present study was undertaken to determine the influence of low intensity microwave radiation (900, 1800, or 2450 MHz for 2h/day, 5days/week, for 60 days) on oxidative stress, inflammatory response and DNA damage in rat brain. Low intensity microwave exposure resulted in a frequency dependent significant increase in oxidative stress markers, reduced levels of GSH and SOD, increased levels of pro-inflammatory cytokines, and significant DNA damage in microwave exposed groups compared to controls. In conclusion, the present study suggests that low intensity microwave radiation induces oxidative stress, inflammatory response and DNA damage in brain by exerting a frequency dependent effect. The study also indicates that increased oxidative stress and inflammatory response might be the factors involved in DNA damage following low intensity microwave exposure.
A series of studies testing different scenarios (called simulations by the study authors) were carried out using incidence data from the Nordic countries to determine the likelihood of detecting various levels of risk as reported in studies of cell phone use and brain tumors between 1979 and 2008. The results were compatible with no increased risks from cell phones, as reported by most epidemiologic studies. The findings did suggest that the increase reported among the subset of heaviest regular users in the Interphone study could not be ruled out but was unlikely. The highly increased risks reported in the Swedish pooled analysis were strongly inconsistent with the observed glioma rates in the Nordic countries (24).
Because of inconsistent findings from epidemiologic studies in humans and the lack of clear data from previous experimental studies in animals, in 1999 the Food and Drug Administration nominated radiofrequency radiation exposure associated with cell phone exposures for study in animal models by the U.S. National Toxicology Program (NTP), an interagency program that coordinates toxicology research and testing across the U.S. Department of Health and Human Services and is headquartered at the National Institute of Environmental Health Sciences, part of NIH.
The RF EME emissions from wi-fi and other wireless devices used for communication are regulated by the Australian Communications and Media Authority (ACMA). The ACMA’s regulatory arrangements require wireless devices to comply with the exposure limits in the ARPANSA RF Standard. The ARPANSA Standard is designed to protect people of all ages and health status against all known adverse health effects from exposure to RF EME. The ARPANSA Standard is based on scientific research that shows the levels at which harmful effects occur and it sets limits, based on international guidelines, well below these harmful levels.
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The study was conducted on 16 adult male Wistar-Albino rats. The rats in the experimental group (n=8) were exposed to 2.4GHz frequency radiation for over a year. The rats in the sham control group (n=8) were subjected to the same experimental conditions except the Wi-Fi generator was turned off. After the exposure period was complete the possible DNA damage on the rat's brain, liver, kidney, skin, and testicular tissues was detected through the single cell gel electrophoresis assay (comet) method. The amount of DNA damage was measured as percentage tail DNA value.
The purpose of the study was to investigate the effect of 2.45 GHz microwave radiation (2 h/day for 35 days) on the male rat brain. MWR-exposed rats showed significantly increased comet head, tail length and tail movement, as well as decrease of antioxidant enzymes. Researchers conclude that the chronic exposure to these radiations may cause significant damage to brain, which may be an indication of possible tumour promotion.
That may be true today. But some experts have grave concerns about the types of low-intensity radiation our wireless devices produce. “We have animal studies suggesting even low-level exposures to the kind of radio wave radiation associated with Wi-Fi could have a variety of negative health effects,” says Joel Moskowitz, director of the Center for Family and Community Health at the University of California, Berkeley. (Moskowitz has collected much of that research here.)
According to a Mar. 2008 meta-analysis of cell phone studies there is a "consistent pattern" connecting cell phone use and an increased risk of developing glioma, a type of brain tumor.  A Mar. 31, 2009 study found that long term cell phone use (10 years +) "approximately doubles the risk" of being diagnosed with glioma on the same side of the head where the cell phone is held.  In Apr. 2013 another study of Swedish cell phone users also found an association between cell phone use and the development of glioma and acoustic neuroma - a benign tumor formation on the nerve near the ear.  That study’s conclusions were confirmed by a different study in Apr. 2014.  Other studies published from 2005-2013 have similarly concluded that there is an association between cell phone use and increased risk of developing brain and head tumors.    
If cell phones were causing cancer we could expect a significant rise in the rate of brain and other related cancers. According to the National Cancer Institute, there was no increase in the incidence of brain or other nervous system cancers between the years 1987 and 2005 despite the fact that cell phone use dramatically increased during those same years.  Between 2004 and 2010 there was still no significant change in the incidence rate of brain tumors. Between 2004 and 2010 there was a slight increase from 209 cases to 221.8 cases per 100,000 people, but this slight increase was attributed to better tracking and recording of cases.  During the same time period, cell phone use increased 62.7% from 182,140,362 subscribers in 2004 to 296,285,629 in 2010. 
The most compelling evidence though comes from a Swedish team of cancer experts whose research stretches back 15 years. The results clearly demonstrate “a consistent association between long-term use of cell phone and cordless phones and glioma and acoustic neuroma”. Overall, they found that using a cell phone for more than a decade significantly increases the risk of a malignant tumour by almost two times with an analogue cell phone and by nearly four times with a digital phone. Interestingly, the risks were even higher for people who had started using mobiles as teenagers.