Thirty-two rats and their forty newborn offspring were divided into the following four groups according to the type of EMR exposure they were subjected to: the control, 900, 1800, and 2450 MHz groups. Each experimental group was exposed to EMR for 60 min/day during the pregnancy and growth periods. The pregnant rats were allowed to stand for four generations (total 52 weeks) before, plasma and uterine samples were obtained. During the 4th, 5th, and 6th weeks of the experiment, plasma and uterine samples were also obtained from the developing rats. [The electric field density was set at 20 dB and 11 V/m in order to obtain 0.1 W/kg whole-body average specific absorption rate (SAR).]
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The most compelling evidence though comes from a Swedish team of cancer experts whose research stretches back 15 years. The results clearly demonstrate “a consistent association between long-term use of cell phone and cordless phones and glioma and acoustic neuroma”. Overall, they found that using a cell phone for more than a decade significantly increases the risk of a malignant tumour by almost two times with an analogue cell phone and by nearly four times with a digital phone. Interestingly, the risks were even higher for people who had started using mobiles as teenagers.
And David Carpenter, M.D., director of the Institute for Health and the Environment at the University at Albany, says that while exposure from a single router in your home may be small, the risks could be greater in places that have dozens of laptops and routers working at the same time—such as school classrooms. Phillips notes that children’s developing bodies may be more vulnerable to all forms of radiation from devices.
We requested semen for analyses from the male patients coming to our infertility division and also asked them to fill out an anonymous questionnaire. We queried their mobile phone and wireless internet usage frequencies in order to determine their radiofrequency-electromagnetic radiation exposure. A total of 1082 patients filled the questionnaire but 51 of them were excluded from the study because of azoospermia.
In conclusion, although EMR exposure decreased the prolactin, estrogen, and progesterone levels in the plasma of maternal rats and their offspring, EMR-induced oxidative stress in the uteri of maternal rats increased during the development of offspring. Mobile phone- and Wi-Fi-induced EMR may be one cause of increased oxidative uterine injury in growing rats and decreased hormone levels in maternal rats. TRPV1 cation channels are the possible molecular pathways responsible for changes in the hormone, oxidative stress, and body temperature levels in the uterus of maternal rats following a year-long exposure to electromagnetic radiation exposure from mobile phones and Wi-Fi devices. It is likely that TRPV1-mediated Ca2+ entry in the uterus of pregnant rats involves accumulation of oxidative stress and opening of mitochondrial membrane pores that consequently leads to mitochondrial dysfunction, substantial swelling of the mitochondria with rupture of the outer membrane and release of oxidants such as superoxide (O2 -) and hydrogen peroxide (H2O2). The superoxide radical is converted to H2O2 by superoxide dismutase (SOD) enzyme. Glutathione peroxidase (GSH-Px) is an important antioxidant enzyme for removing lipid hydroperoxides and hydrogen peroxide and it catalyzes the reduction of H2O2 to water.
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It is useful to be aware of new health research regarding cell phone usage and cell phone radiation. The first cell phone call was made in 1985 and that phone cost $5,000 and weighed about 9 pounds. The change in size, weight and cost of devices today has probably led to over 50% of the human race owning a mobile device, the fastest growing technology on the planet.